Serum and tissue 2HVEM3 levels in patients with multiple or recurrent verruca vulgaris /
Marwa Abdelghany Mansour
Serum and tissue 2HVEM3 levels in patients with multiple or recurrent verruca vulgaris / مستوى مادة "الهيفم " في المصل والجلد للمرضى المصابين بالثآليل الفيروسية المتعددة أو المتكررة Marwa Abdelghany Mansour ; Supervised Manal A-W Bosseila , Olfat G. Shaker , Dina M. Kadry - Cairo : Marwa Abdelghany Mansour , 2015 - 114 P. : charts ; 25cm
Thesis (M.Sc.) - Cairo University - Faculty of Medicine - Department of Dermatology and Venereology
Warts are benign lesions caused by human papilloma virus (HPV). Host defense against HPV relies on intact and functioning cellular immunity including T cell and natural killer (NK) cell cytotoxicity. Therefore, in patients in whom warts are severe, concern for immune defects is raised. HVEM (herpes virus entry mediator) is a member of the tumour-necrosis factor receptor family. It has diverse functions, augmenting or inhibiting the immune response. It acts as a receptor to enhance immune responses through binding LIGHT or LTalpha which signaling is important for cell survival, inflammation, and tumor eradication. Aim of work: To identify the role of cell-mediated immunity, expressed as HVEM level, in cases of multiple/recurrent verrucae, in order to assess its role in the adaptive immune response to HPV
HVEM Serum Tissue
Serum and tissue 2HVEM3 levels in patients with multiple or recurrent verruca vulgaris / مستوى مادة "الهيفم " في المصل والجلد للمرضى المصابين بالثآليل الفيروسية المتعددة أو المتكررة Marwa Abdelghany Mansour ; Supervised Manal A-W Bosseila , Olfat G. Shaker , Dina M. Kadry - Cairo : Marwa Abdelghany Mansour , 2015 - 114 P. : charts ; 25cm
Thesis (M.Sc.) - Cairo University - Faculty of Medicine - Department of Dermatology and Venereology
Warts are benign lesions caused by human papilloma virus (HPV). Host defense against HPV relies on intact and functioning cellular immunity including T cell and natural killer (NK) cell cytotoxicity. Therefore, in patients in whom warts are severe, concern for immune defects is raised. HVEM (herpes virus entry mediator) is a member of the tumour-necrosis factor receptor family. It has diverse functions, augmenting or inhibiting the immune response. It acts as a receptor to enhance immune responses through binding LIGHT or LTalpha which signaling is important for cell survival, inflammation, and tumor eradication. Aim of work: To identify the role of cell-mediated immunity, expressed as HVEM level, in cases of multiple/recurrent verrucae, in order to assess its role in the adaptive immune response to HPV
HVEM Serum Tissue