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A study of T- regulatory cells as a possible marker of severity in bronchial asthma / Heba Allah Ahmed Moussa ; Supervised Esmat Ali Abdelnabi , Sherif Nasseh Amin , Mostafa Ibrahim Elshazly

By: Contributor(s): Material type: TextTextLanguage: eng Publication details: Cairo : Heba Allah Ahmed Moussa , 2010Description: 171P. : charts , facsimiles ; 25cmOther title:
  • دراسة خلايا "تى" الليمفاوية المنظمة كدلالة محتملة لتقييم شدة مرض الربو الشعبى [Added title page title]
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Dissertation note: Thesis (Ph.D.) - Cairo University - Faculty of Medicine - Department of Chest diseases and Tuberculosis Summary: Asthma is chronic inflammatory disease of the lung . It occurs as a result of inappropriate type 2 - dominated immune response to environmental allergens . Cytokines (IL4, IL5 and IL13) have been shown to regulate the pathological features of the disease including airway hyper - responsiveness (AHR) eosinophilic pulmonary inflammation airway remodeling and IgE synthesis
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Item type Current library Home library Call number Copy number Status Barcode
Thesis Thesis قاعة الرسائل الجامعية - الدور الاول المكتبة المركزبة الجديدة - جامعة القاهرة Cai01.11.06.Ph.D.2010.He.S (Browse shelf(Opens below)) Not for loan 01010110053066000
CD - Rom CD - Rom مخـــزن الرســائل الجـــامعية - البدروم المكتبة المركزبة الجديدة - جامعة القاهرة Cai01.11.06.Ph.D.2010.He.S (Browse shelf(Opens below)) 53066.CD Not for loan 01020110053066000

Thesis (Ph.D.) - Cairo University - Faculty of Medicine - Department of Chest diseases and Tuberculosis

Asthma is chronic inflammatory disease of the lung . It occurs as a result of inappropriate type 2 - dominated immune response to environmental allergens . Cytokines (IL4, IL5 and IL13) have been shown to regulate the pathological features of the disease including airway hyper - responsiveness (AHR) eosinophilic pulmonary inflammation airway remodeling and IgE synthesis

Issued also as CD

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