Antifibrotic potential of a selective COX-2 inhibitor (celecoxib) and a synthetic PPAR y ligand (pioglitazone) on liver fibrosis in Rats / Noha Samir Abdellatif ; Supervised Ahmed Hussein Galal , Fatma Ahmed Elbatrawi , Essam Fouad Alalkamy

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Noha Samir Abdellatif

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TextLanguage: English Publication details: Cairo : Noha Samir Abdellatif , 2011Description: 154 P. : charts , facsimiles ; 25cmOther title:

التأثير المحتمل لمثبط كوكس ٢ الإنتقائى (سيليكوكسيب) و منشط ببار جاما الإصطناعي (بيوجليتازون) علي تليف الكبد في الجرذان [Added title page title]

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Dissertation note: Thesis (M.Sc.) - Cairo University - Faculty of Medicine - Department of Pharmacology Summary: Chronic liver injury, independent of the cause, is generally associated with the accumulation of matrix proteins, a process referred to as fibrosis. Over the last decade liver fibrosis is no longer viewed as either passive or permanent but as a dynamic process and potential therapies continue to be identified. Hepatic stellate cells (HSCs) play a critical role in the fibrogenesis of liver, so they are the target cells of antifibrotic therapy

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Item type	Current library	Home library	Call number	Copy number	Status	Barcode
Thesis	قاعة الرسائل الجامعية - الدور الاول	المكتبة المركزبة الجديدة - جامعة القاهرة	Cai01.11.29.M.Sc.2011.No.A (Browse shelf(Opens below))		Not for loan	01010110055774000
CD - Rom	مخـــزن الرســائل الجـــامعية - البدروم	المكتبة المركزبة الجديدة - جامعة القاهرة	Cai01.11.29.M.Sc.2011.No.A (Browse shelf(Opens below))	55774.CD	Not for loan	01020110055774000

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Thesis (M.Sc.) - Cairo University - Faculty of Medicine - Department of Pharmacology

Chronic liver injury, independent of the cause, is generally associated with the accumulation of matrix proteins, a process referred to as fibrosis. Over the last decade liver fibrosis is no longer viewed as either passive or permanent but as a dynamic process and potential therapies continue to be identified. Hepatic stellate cells (HSCs) play a critical role in the fibrogenesis of liver, so they are the target cells of antifibrotic therapy

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