The relation between plasma concentration of osteopontin and matrix metalloproteinase-2 and severity of chronic heart failure / Alaa Abdalhamid Mohammed Ali ; Supervised Amina Kamal Eldein Elansary , Yaser Ahmed Abdelhady , Salwa Fayez Hasan

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Alaa Abdalhamid Mohammed Ali

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TextLanguage: English Publication details: Cairo : Alaa Abdalhamid Mohammed Ali , 2012Description: 155 P. : charts , facsimiles ; 25cmOther title:

العلاقة بينتركيز الأوستيوبونتين و الميتالوبروتينيز-2 فى البلازما و شدة فشل عضلة القلب المزمن [Added title page title]

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Dissertation note: Thesis (Ph.D.) - Cairo University - Faculty of Medicine - Department of Medical Biochemistry Summary: Extracellular matrix remodeling is thought to play an important role in the progression of heart failure (HF). Matrix metalloproteinases-2 (MMPs) havebeen demonstrated to influence left ventricular properties and serve as targets of potential anti-remodeling agents. osteopontin, a glycoprotein that can be detected in plasma, was found to be up regulated in several animal models of cardiac failure and may thus represent a new biomarker that facilitates risk stratification in patients with heart failure

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Item type	Current library	Home library	Call number	Copy number	Status	Date due	Barcode
Thesis	قاعة الرسائل الجامعية - الدور الاول	المكتبة المركزبة الجديدة - جامعة القاهرة	Cai01.11.03.Ph.D.2012.Al.R (Browse shelf(Opens below))		Not for loan		01010110058935000
CD - Rom	مخـــزن الرســائل الجـــامعية - البدروم	المكتبة المركزبة الجديدة - جامعة القاهرة	Cai01.11.03.Ph.D.2012.Al.R (Browse shelf(Opens below))	58935.CD	Not for loan		01020110058935000

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Thesis (Ph.D.) - Cairo University - Faculty of Medicine - Department of Medical Biochemistry

Extracellular matrix remodeling is thought to play an important role in the progression of heart failure (HF). Matrix metalloproteinases-2 (MMPs) havebeen demonstrated to influence left ventricular properties and serve as targets of potential anti-remodeling agents. osteopontin, a glycoprotein that can be detected in plasma, was found to be up regulated in several animal models of cardiac failure and may thus represent a new biomarker that facilitates risk stratification in patients with heart failure

Issued also as CD

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