Assessment of the effect of pyrroloquinoline quinone and resveratrol on the survival and regeneration of cerebellar granular neurons / Nagwan Nabil Hussien Ibrahim Shanan ; Supervised Hans Georg Breitinger , Reham Mahmoud Abdelkader

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Nagwan Nabil Hussien Ibrahim Shanan

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Material type: Text

TextLanguage: English Publication details: Cairo : Nagwan Nabil Hussien Ibrahim Shanan , 2015Description: 123 Leaves : charts , facsimiles , photographs , maps ; 30cmSubject(s):

Dissertation note: Thesis (Ph.D.) - German University - Faculty of Postgraduate Studies and Scientific Research - Department of Pharmacology and Toxicology Summary: Neuronal damage and degeneration are key events following stroke, and injuries to the brain and spinal cord. Under such critical conditions, degeneration neurons do not have the ability to regenerate and their capacity to re-establish lost connections is limited, which can lead to long term disability. The inability of central nervous system (CNS) neurons to regenerate following injury can be attributed to several factors including (i) loss of cells due to apoptosis, (ii) the extrinsic inhibitory environment, (iii) the limited intrinsic potential of neurons to regenerate. Experimental evidence has shown that fostering the intrinsic potental of damaged neurons to regenerate and sprout may be a promising therapeutic approach

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Item type	Current library	Home library	Call number	Status	Barcode
Thesis	قاعة الثقاقات الاجنبية - الدور الثالث	المكتبة المركزبة الجديدة - جامعة القاهرة	Cai01.34.Ph.D.2015.Na.A (Browse shelf(Opens below))	Not for loan	01010110072263000

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Thesis (Ph.D.) - German University - Faculty of Postgraduate Studies and Scientific Research - Department of Pharmacology and Toxicology

Neuronal damage and degeneration are key events following stroke, and injuries to the brain and spinal cord. Under such critical conditions, degeneration neurons do not have the ability to regenerate and their capacity to re-establish lost connections is limited, which can lead to long term disability. The inability of central nervous system (CNS) neurons to regenerate following injury can be attributed to several factors including (i) loss of cells due to apoptosis, (ii) the extrinsic inhibitory environment, (iii) the limited intrinsic potential of neurons to regenerate. Experimental evidence has shown that fostering the intrinsic potental of damaged neurons to regenerate and sprout may be a promising therapeutic approach

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