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Role of dexmedetomidine in modifying immune paralysis in patient with septic shock : Randomized cotrolled trial (RODI Strial) / Eman Ahmed Ahmed ; Suervised Ahmed Mohamed Mukhtar , Mohamed Elayashy Mohamed , Sahar Mahmoud Mohamed Kasem

By: Contributor(s): Material type: TextTextLanguage: English Publication details: Cairo : Eman Ahmed Ahmed , 2021Description: 56 P. : charts , facsimiles ; 25cmOther title:
  • دور عقار الديكسميديتوميدين فى تعديل الشلل المناعى للمرضى المصابين بصدمه انتنانيه : تجربة عشوائيه محكمه [Added title page title]
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Dissertation note: Thesis (Ph.D.) - Cairo University - Faculty of Medicine - Department of Anaesthesia Summary: The condition of insusceptible loss of motion is caused essentially by lessened capacity of monocytes to deliver favorable to inflammatory cytokines in light of endotoxin.This wonder is known as endotoxin resilience (1). The impact of endotoxin resilience on leuckocytes causes an increment in the arrival of immunosuppressive middle people for the most part interleukin-10 (IL-10 and a reduction in antigen show because of diminished articulation of human leukocyte antigen-DR (HLADR). Besides, there is an advancing proof of platelet commitment in inflammation and early reaction of the host safeguard.(2) Following actuation, platelet-inferred microparticles
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Item type Current library Home library Call number Copy number Status Date due Barcode
Thesis Thesis قاعة الرسائل الجامعية - الدور الاول المكتبة المركزبة الجديدة - جامعة القاهرة Cai01.11.01.Ph.D.2021.Em.R (Browse shelf(Opens below)) Not for loan 01010110085480000
CD - Rom CD - Rom مخـــزن الرســائل الجـــامعية - البدروم المكتبة المركزبة الجديدة - جامعة القاهرة Cai01.11.01.Ph.D.2021.Em.R (Browse shelf(Opens below)) 85480.CD Not for loan 01020110085480000

Thesis (Ph.D.) - Cairo University - Faculty of Medicine - Department of Anaesthesia

The condition of insusceptible loss of motion is caused essentially by lessened capacity of monocytes to deliver favorable to inflammatory cytokines in light of endotoxin.This wonder is known as endotoxin resilience (1). The impact of endotoxin resilience on leuckocytes causes an increment in the arrival of immunosuppressive middle people for the most part interleukin-10 (IL-10 and a reduction in antigen show because of diminished articulation of human leukocyte antigen-DR (HLADR). Besides, there is an advancing proof of platelet commitment in inflammation and early reaction of the host safeguard.(2) Following actuation, platelet-inferred microparticles

Issued also as CD

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