| 000 | 01777cam a2200313 a 4500 | ||
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| 003 | EG-GiCUC | ||
| 008 | 131028s2013 ua dh f m 000 0 eng d | ||
| 040 |
_aEG-GiCUC _beng _cEG-GiCUC |
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| 041 | 0 | _aeng | |
| 049 | _aDeposite | ||
| 097 | _aM.Sc | ||
| 099 | _aCai01.08.06.M.Sc.2013.Ma.P | ||
| 100 | 0 | _aMai Muhammed Ragab | |
| 245 | 1 | 0 |
_aProtein kinase R (PKR) expression as a predictive factor for interferon response in chronic hepatitis C patients / _cMai Muhammed Ragab ; Supervised Magdy Ali Amin , Amal Ahmed Mohamed |
| 246 | 1 | 5 | _aنسبة وجود جين بروتين كايناز كدليل على مدى استجابة مرضى الالتهاب الكبدى الوبائى المزمن سى للعلاج بالانترفيرون |
| 260 |
_aCairo : _bMai Muhammed Ragab , _c2013 |
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| 300 |
_a99 P. : _bcharts , facsimiles ; _c25cm |
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| 502 | _aThesis (M.Sc.) - Cairo University - Faculty of Pharmacy - Department of Microbiology and Immunology | ||
| 520 | _aEgypt has the highest prevalence of hepatitis C virus (HCV) worldwide . Currently combined pegylated interferon and ribavirin therapy are the standard treatment . The biological activity of interferon (IFN) is mediated by the induction of intracellular antiviral proteins, such as 2'-5' oligoadenylate synthetase, and dsRNA - activated protein kinase. IFN inducible double - stranded RNA- activated protein kinase (PKR) is thought to play a key antiviral role against HCV | ||
| 530 | _aIssued also as CD | ||
| 653 | 4 | _aChronic hepatitis C | |
| 653 | 4 | _aPegylated interfern | |
| 653 | 4 | _aProtein kinase gene | |
| 700 | 0 |
_aAmal Ahmed Mohamed , _eSupervisor |
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| 700 | 0 |
_aMagdy Ali Amin , _eSupervisor |
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| 905 |
_aNazla _eRevisor |
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| 905 |
_aSoheir _eCataloger |
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| 942 |
_2ddc _cTH |
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| 999 |
_c43914 _d43914 |
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