000			02057cam a2200349 a 4500
003			EG-GiCUC
005			20250223031047.0
008			140929s2013 ua dh f m 000 0 eng d
040			_aEG-GiCUC _beng _cEG-GiCUC
041	0		_aeng
049			_aDeposite
097			_aM.Sc
099			_aCai01.11.03.M.Sc.2013.En.I
100	0		_aEngy Medhat Ahmed Abdelmegeed
245	1	0	_aInterleukin - 10 gene polymorphisms in type - 2 diabetic patients with nephropathy / _cEngy Medhat Ahmed Abdelmegeed ; Supervised Samia Mostafa Mohamed , Manal Nabih Eldeeb , Nermeen Mohamed Hassan
246	1	5	_aالتحور الجينى للانترلوكين - 10 في مرضي الاعتلال الكلوى الناتج عن النوع الثانى للبوال السكرى
260			_aCairo : _bEngy Medhat Ahmed Abdelmegeed , _c2013
300			_a108 P. : _bcharts , facsimiles ; _c25cm
502			_aThesis (M.Sc.) - Cairo University - Faculty of Medicine - Department of Medical Biochemistry
520			_aDiabetic nephropathy is the most prevalent cause for end - stage renal disease in the united states and the western world. In this disease, tubular injury and progressive interstitial fibrosis contribute significantly to renal failure and predict progression to end-stage renal disease. In early diabetic renal injury, there is podocyte drop - out which is thought to cause glomerular proteinuria and subsequent diabetic glomerular injury. Different inflammatory molecules including pro - inflammatory cytokines have been proposed as critical factors in the development of microvascular diabetic complications including nephropathy
530			_aIssued also as CD
653		4	_aDiabetic nephropathy
653		4	_aInterleukin - 10
653		4	_aType 2 diabetes
700	0		_aManal Nabih Eldeeb , _eSupervisor
700	0		_aNermeen Mohamed Hassan , _eSupervisor
700	0		_aSamia Mostafa Mohamed , _eSupervisor
856			_uhttp://172.23.153.220/th.pdf
905			_aNazla _eRevisor
905			_aSamia _eCataloger
942			_2ddc _cTH
999			_c47590 _d47590