| 000 | 01550cam a2200301 a 4500 | ||
|---|---|---|---|
| 003 | EG-GiCUC | ||
| 008 | 170812s2014 ua dh f m 000 0 eng d | ||
| 040 |
_aEG-GiCUC _beng _cEG-GiCUC |
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| 041 | 0 | _aeng | |
| 049 | _aGift | ||
| 097 | _aM.Sc | ||
| 099 | _aCai01.34.M.Sc.2014.Mo.M | ||
| 100 | 0 | _aMohammad Mamdouh Abdelwareth | |
| 245 | 1 | 0 |
_aMolecular repair mechanisms-combined effects of gain-and loss-of- function mutants on channel function of the inhibitory glycine receptor / _cMohammad Mamdouh Abdelwareth ; Supervised Hans-Georg Breitinger , Carmen Villmann , Ulrike Breitinger |
| 260 |
_aCairo : _bMohammad Mamdouh Abdelwareth , _c2014 |
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| 300 |
_a98 Leaves : _bcharts , facsimiles ; _c30cm |
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| 502 | _aThesis (M.Sc.) - Garman University - Faculty of Postgraduate Studies and Scientific Research - Department of Biotechnology | ||
| 520 | _aFast synaptic inhibitory neurotransmission in the central nervous system is principally mediated by {u01B4}-aminobutyric acid type A receptors(GABAAR) and glycine receptors(G1yR). The spinal cord and brainstem are main sites of actionof glycine receptors. Glycinergic synapses are present in spinal cord motor reflex pathways, spinal cord nociceptive ( pain sensory) pathawys. | ||
| 653 | 4 | _aChannel function | |
| 653 | 4 | _aGlycine | |
| 653 | 4 | _aSynapse | |
| 700 | 0 |
_aCarmen Villmann , _esupervisor |
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| 700 | 0 |
_aHans-Georg Breitinger , _esupervisor |
|
| 700 | 0 |
_aUlrike Breitinger , _esupervisor |
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| 905 |
_aNazla _eRevisor |
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| 905 |
_aShimaa _eCataloger |
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| 942 |
_2ddc _cTH |
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| 999 |
_c61923 _d61923 |
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