000 | 02073cam a2200337 a 4500 | ||
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003 | EG-GiCUC | ||
008 | 220226s2021 ua dh f m 000 0 eng d | ||
040 |
_aEG-GiCUC _beng _cEG-GiCUC |
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041 | 0 | _aeng | |
049 | _aDeposite | ||
097 | _aPh.D | ||
099 | _aCai01.11.01.Ph.D.2021.Em.R | ||
100 | 0 | _aEman Ahmed Ahmed | |
245 | 1 | 0 |
_aRole of dexmedetomidine in modifying immune paralysis in patient with septic shock : _bRandomized cotrolled trial (RODI Strial) / _cEman Ahmed Ahmed ; Suervised Ahmed Mohamed Mukhtar , Mohamed Elayashy Mohamed , Sahar Mahmoud Mohamed Kasem |
246 | 1 | 5 |
_aدور عقار الديكسميديتوميدين فى تعديل الشلل المناعى للمرضى المصابين بصدمه انتنانيه : _bتجربة عشوائيه محكمه |
260 |
_aCairo : _bEman Ahmed Ahmed , _c2021 |
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300 |
_a56 P. : _bcharts , facsimiles ; _c25cm |
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502 | _aThesis (Ph.D.) - Cairo University - Faculty of Medicine - Department of Anaesthesia | ||
520 | _aThe condition of insusceptible loss of motion is caused essentially by lessened capacity of monocytes to deliver favorable to inflammatory cytokines in light of endotoxin.This wonder is known as endotoxin resilience (1). The impact of endotoxin resilience on leuckocytes causes an increment in the arrival of immunosuppressive middle people for the most part interleukin-10 (IL-10 and a reduction in antigen show because of diminished articulation of human leukocyte antigen-DR (HLADR). Besides, there is an advancing proof of platelet commitment in inflammation and early reaction of the host safeguard.(2) Following actuation, platelet-inferred microparticles | ||
530 | _aIssued also as CD | ||
650 | 0 | _aSeptic shock | |
653 | _aDexmedetomidine | ||
653 | _aImmune paralysis | ||
653 | _aModifying | ||
700 | 0 |
_aAhmed Mohamed Mukhtar , _eSupervisor |
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700 | 0 |
_aMohamed Elayashy Mohamed , _eSupervisor |
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700 | 0 |
_aSahar Mahmoud Mohamed Kasem , _eSupervisor |
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905 |
_aNazla _eRevisor |
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905 |
_aShimaa _eCataloger |
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942 |
_2ddc _cTH |
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999 |
_c84333 _d84333 |