000 02073cam a2200337 a 4500
003 EG-GiCUC
008 220226s2021 ua dh f m 000 0 eng d
040 _aEG-GiCUC
_beng
_cEG-GiCUC
041 0 _aeng
049 _aDeposite
097 _aPh.D
099 _aCai01.11.01.Ph.D.2021.Em.R
100 0 _aEman Ahmed Ahmed
245 1 0 _aRole of dexmedetomidine in modifying immune paralysis in patient with septic shock :
_bRandomized cotrolled trial (RODI Strial) /
_cEman Ahmed Ahmed ; Suervised Ahmed Mohamed Mukhtar , Mohamed Elayashy Mohamed , Sahar Mahmoud Mohamed Kasem
246 1 5 _aدور عقار الديكسميديتوميدين فى تعديل الشلل المناعى للمرضى المصابين بصدمه انتنانيه :
_bتجربة عشوائيه محكمه
260 _aCairo :
_bEman Ahmed Ahmed ,
_c2021
300 _a56 P. :
_bcharts , facsimiles ;
_c25cm
502 _aThesis (Ph.D.) - Cairo University - Faculty of Medicine - Department of Anaesthesia
520 _aThe condition of insusceptible loss of motion is caused essentially by lessened capacity of monocytes to deliver favorable to inflammatory cytokines in light of endotoxin.This wonder is known as endotoxin resilience (1). The impact of endotoxin resilience on leuckocytes causes an increment in the arrival of immunosuppressive middle people for the most part interleukin-10 (IL-10 and a reduction in antigen show because of diminished articulation of human leukocyte antigen-DR (HLADR). Besides, there is an advancing proof of platelet commitment in inflammation and early reaction of the host safeguard.(2) Following actuation, platelet-inferred microparticles
530 _aIssued also as CD
650 0 _aSeptic shock
653 _aDexmedetomidine
653 _aImmune paralysis
653 _aModifying
700 0 _aAhmed Mohamed Mukhtar ,
_eSupervisor
700 0 _aMohamed Elayashy Mohamed ,
_eSupervisor
700 0 _aSahar Mahmoud Mohamed Kasem ,
_eSupervisor
905 _aNazla
_eRevisor
905 _aShimaa
_eCataloger
942 _2ddc
_cTH
999 _c84333
_d84333