Role of Pentraxin 3 and Doppler ultrasound in assessing the severity of endothelial dysfunction in patients on regular hemodialysis in comparison with chronic kidney disease patients stage 5 not on renal replacement therapy / (Record no. 178340)

MARC details
000 -LEADER
fixed length control field 06927namaa22004571i 4500
003 - CONTROL NUMBER IDENTIFIER
control field EG-GICUC
005 - أخر تعامل مع التسجيلة
control field 20260219110917.0
008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION
fixed length control field 260210s2024 ua a|||frm||| 000 0 eng d
040 ## - CATALOGING SOURCE
Original cataloguing agency EG-GICUC
Language of cataloging eng
Transcribing agency EG-GICUC
Modifying agency EG-GICUC
Description conventions rda
041 0# - LANGUAGE CODE
Language code of text/sound track or separate title eng
Language code of summary or abstract eng
-- ara
049 ## - Acquisition Source
Acquisition Source Deposit
082 04 - DEWEY DECIMAL CLASSIFICATION NUMBER
Classification number 616.61
092 ## - LOCALLY ASSIGNED DEWEY CALL NUMBER (OCLC)
Classification number 616.61
Edition number 21
097 ## - Degree
Degree Ph.D
099 ## - LOCAL FREE-TEXT CALL NUMBER (OCLC)
Local Call Number Cai01.11.18.Ph.D.2024.Ba.R
100 0# - MAIN ENTRY--PERSONAL NAME
Authority record control number or standard number Basma Abd El-Aziem Zaghloul Mohamed,
Preparation preparation.
245 10 - TITLE STATEMENT
Title Role of Pentraxin 3 and Doppler ultrasound in assessing the severity of endothelial dysfunction in patients on regular hemodialysis in comparison with chronic kidney disease patients stage 5 not on renal replacement therapy /
Statement of responsibility, etc. by Basma Abd El-Aziem Zaghloul Mohamed ; Supervised Prof. Mohamed Mostafa Elkhati, Prof. Amr Mohamed Shaker, Dr. Heba Mahmoud Ibrahim, Prof. Tarek Ramzy Abd El-Aziem.
246 15 - VARYING FORM OF TITLE
Title proper/short title دور البنتراكسين 3 والدوبلر في تقييم شدة الخلل البطاني لدى مرضى الغسيل الكلى المنتظم مقارنة مع مرضى القصور الكلوي المزمن في المرحلة الخامسة الذين لا يخضعون لغسيل الكلى
264 #0 - PRODUCTION, PUBLICATION, DISTRIBUTION, MANUFACTURE, AND COPYRIGHT NOTICE
Date of production, publication, distribution, manufacture, or copyright notice 2024.
300 ## - PHYSICAL DESCRIPTION
Extent 130 pages :
Other physical details illustrations ;
Dimensions 25 cm. +
Accompanying material CD.
336 ## - CONTENT TYPE
Content type term text
Source rda content
337 ## - MEDIA TYPE
Media type term Unmediated
Source rdamedia
338 ## - CARRIER TYPE
Carrier type term volume
Source rdacarrier
502 ## - DISSERTATION NOTE
Dissertation note Thesis (Ph.D)-Cairo University, 2025.
504 ## - BIBLIOGRAPHY, ETC. NOTE
Bibliography, etc. note Bibliography: pages 100-129.
520 #3 - SUMMARY, ETC.
Summary, etc. Background: Endothelial dysfunction (ED) is a major contributor to the pro-<br/>inflammatory state, as well as a leading cause of subclinical atherosclerosis and <br/>consequently, cardiovascular disease, which is primary cause of death in end stage <br/>renal disease population. Pentraxin3 (PTX3) is a potential biomarker of vascular <br/>inflammation in addition to measuring Flow mediated dilatation of brachial artery <br/>(FMD)%, and carotid intima media thickness (CIMT) are noninvasive techniques in <br/>assessing endothelial dysfunction. <br/>Aim: <br/>Evaluating the significance of serum PTX 3, FMD%, and CIMT in identifying en-<br/>dothelial dysfunction in patients with stage 5 chronic kidney disease who are not r-<br/>eceiving renal replacement therapy in comparison to hemodialysis patients. <br/>Methods: a prospective cross-sectional study with 150 individuals overall, <br/>classified into 3 groups: 50 individuals for each group: i) Control group; ii) CKD <br/>group (stage 5 not on renal replacement therapy); and iii) Dialysis group. All of them <br/>were subjected to clinical assessment, laboratory measurements of lipid profile, <br/>calcium, phosphorus, PTH as well as serum PTX3. Additionally, Doppler <br/>ultrasonography was used to assess CIMT, FMD%, and flow volume. <br/>Results: According to our results, PTX3 level was significantly higher in CKD and <br/>dialysis patients compared to controls; moreover, its level is higher in dialysis <br/>patients compared to CKD with p <0.001. The FMD% and its flow volume were <br/>significantly lower, while the CIMT was significantly higher in CKD and dialysis <br/>patients compared to controls; furthermore, the FMD% and its flow volume were <br/>lower in CKD compared to dialysis patients, with a p-value of <0.001.PXT3 showed <br/>significant negative correlation with FMD% and its flow volume and positive <br/>correlation with CIMT. Based on the suggested cut-off on ROC curve and its area <br/>under curve, PTX3, FMD% had a high sensitivity (82% and 80% respectively) and <br/>high specificity (75%, and 85% respectively) <br/>Conclusion: High levels of PTX3, low FMD%, and increased CIMT play a crucial <br/>role in assessing endothelial dysfunction in CKD and dialysis patients. Furthermore, <br/>we hypothesized that dialysis may improve the endothelial dysfunction, which is <br/>confirmed by higher PTX3 and lower FMD% in CKD compared to dialysis patients.
520 #3 - SUMMARY, ETC.
Summary, etc. يعد القصورالكلوي المزمن مشكلة صحية عالمية كبرى تؤثر على أكثر من 13% من البالغين في العالم. في مرضى الكلى المزمن ،و تمثل مرض القلب والأوعية الدموية السبب الرئيسي للحالات المرضية المصاحبة للقصور الكلوي وسبب الوفاة الأكثر أهميةز من الفشل الكلوي نفسه. يلعب خلل بطانة الأوعية الدموية دورًا مهمًا في نشوء تغيرات فسيولوجية متعددة و ارتفاع نسبة المواد المعززة للالتهابات التي تزيد في حالات القصور الكلوي و التي تساهم بشكل اساسي في حدوث امراض القلب و الأوعية الدموية. <br/>لقد برز قياس وظيفة بطانة الأوعية الدموية كأداة قيمة تعكس التغيرات الفسيولوجية التي تؤدي الي حدوث امراض القلب و الأوعية الدموية و قد تتنبأ بحدوث هذه الامراض بشكل مبكر . إن قياس التمدد الناتج عن التدفق هو نهج غير تداخلي يعكس وظيفة الشرايين المعتمدة على بطانة الأوعية الدموية والتي تعتمد إلى حد كبير على أكسيد النيتريك وقد تم استخدامه كأداة قياس لصحة الأوعية الدموية. علاوة على ذلك، فإن قياس إن تقييم التمدد الناتج عن التدفق هو أداة لفحص الفيسيولوجيا المرضية للأمراض القلبية الوعائية وربما تحديد الأشخاص المعرضين لخطر متزايد لأمراض القلب و الأوعية الدموية. و يلعب البنتراكسين 3 دورًا مهما في الاستجابة المناعية و له تأثير كوسيط لحدوث تغيرات تؤثر على فسيولوجيا نظام القلب والأوعية الدموية من خلال مجموعة متنوعة من الظواهر مثل الالتهاب، وتولد الأوعية، وتكون الأورام، والتصاق الخلايا..
530 ## - ADDITIONAL PHYSICAL FORM AVAILABLE NOTE
Issues CD Issues also as CD.
546 ## - LANGUAGE NOTE
Text Language Text in English and abstract in Arabic & English.
650 #0 - SUBJECT ADDED ENTRY--TOPICAL TERM
Topical term or geographic name entry element Internal Medicine
650 #0 - SUBJECT ADDED ENTRY--TOPICAL TERM
Topical term or geographic name entry element أمراض الكلى
653 #1 - INDEX TERM--UNCONTROLLED
Uncontrolled term pentraxin
-- FMD
-- carotid intima media thickness
-- chronic kidney disease
700 0# - ADDED ENTRY--PERSONAL NAME
Personal name Mohamed Mostafa Elkhati
Relator term thesis advisor.
700 0# - ADDED ENTRY--PERSONAL NAME
Personal name Amr Mohamed Shaker
Relator term thesis advisor.
700 0# - ADDED ENTRY--PERSONAL NAME
Personal name Heba Mahmoud Ibrahim,
Relator term thesis advisor.
700 0# - ADDED ENTRY--PERSONAL NAME
Personal name Tarek Ramzy Abd El-Aziem
Relator term thesis advisor.
900 ## - Thesis Information
Grant date 01-01-2025
Supervisory body Mohamed Mostafa Elkhati
-- Amr Mohamed Shaker
-- Heba Mahmoud Ibrahim
-- Tarek Ramzy Abd El-Aziem
Universities Cairo University
Faculties Faculty of Medicine
Department Department of Internal Medicine
905 ## - Cataloger and Reviser Names
Cataloger Name Shimaa
Reviser Names Eman Ghareb
942 ## - ADDED ENTRY ELEMENTS (KOHA)
Source of classification or shelving scheme Dewey Decimal Classification
Koha item type Thesis
Edition 21
Suppress in OPAC No
Holdings
Source of classification or shelving scheme Home library Current library Date acquired Inventory number Full call number Barcode Date last seen Effective from Koha item type
Dewey Decimal Classification المكتبة المركزبة الجديدة - جامعة القاهرة قاعة الرسائل الجامعية - الدور الاول 10.02.2026 93338 Cai01.11.18.Ph.D.2024.Ba.R 01010110093338000 10.02.2026 10.02.2026 Thesis
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