Impact of notch1 signaling pathway on clinical course of pediatric t-cell acute lymphoblastic leukemia / (رقم التسجيلة. 84372)

تفاصيل مارك
000 -LEADER
fixed length control field 04062nam a2200409 a 4500
003 - CONTROL NUMBER IDENTIFIER
control field EG-GiCUC
005 - أخر تعامل مع التسجيلة
control field 20260516142557.0
008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION
fixed length control field 220228s2021 ua dh frm 000 0 eng d
040 ## - CATALOGING SOURCE
Original cataloguing agency EG-GiCUC
Transcribing agency EG-GiCUC
Language of cataloging eng
041 0# - LANGUAGE CODE
Language code of text/sound track or separate title eng
Language code of summary or abstract eng
-- ara
049 ## - Acquisition Source
Acquisition Source Deposite
082 04 - DEWEY DECIMAL CLASSIFICATION NUMBER
Classification number 540
092 ## - LOCALLY ASSIGNED DEWEY CALL NUMBER (OCLC)
Classification number 540
Edition number 21
097 ## - Degree
Degree M.Sc
099 ## - LOCAL FREE-TEXT CALL NUMBER (OCLC)
Local Call Number Cai01.12.25.M.Sc.2022.Ma.I
100 0# - MAIN ENTRY--PERSONAL NAME
Authority record control number or standard number Manar Hesham Fouad,
Preparation preparation.
245 10 - TITLE STATEMENT
Title Impact of notch1 signaling pathway on clinical course of pediatric t-cell acute lymphoblastic leukemia /
Statement of responsibility, etc. by Manar Hesham Fouad ; Supervised Prof.Dr.Mahfouz Ali Abdelaziz , Prof.Dr.Nashwa Nagy Elkhazragy.
246 15 - VARYING FORM OF TITLE
Title proper/short title تأثير مسار نوتش على خط السير الاكلينكي لمرض سرطان الدم اللميفاوي الحاد من نوع الخلية التائية فى الأطفال
264 #0 - PRODUCTION, PUBLICATION, DISTRIBUTION, MANUFACTURE, AND COPYRIGHT NOTICE
Date of production, publication, distribution, manufacture, or copyright notice 2022
300 ## - PHYSICAL DESCRIPTION
Extent 216 Pages :
Other physical details charts , facsimiles ;
Dimensions 30 cm+
Accompanying material CD
336 ## - CONTENT TYPE
Content type term text
Source rda content
337 ## - MEDIA TYPE
Media type term Unmediated
Source rdamedia
338 ## - CARRIER TYPE
Carrier type term volume
Source rdacarrier
502 ## - DISSERTATION NOTE
Dissertation note Thesis (M.Sc.) - Cairo University - Faculty of Science - Department of Organic Chemistry
504 ## - BIBLIOGRAPHY, ETC. NOTE
Bibliography, etc. note Bibliography: pages 140-227.
520 #3 - SUMMARY, ETC.
Summary, etc. Acute Lymphoblastic Leukemia (ALL) represents the most commonly diagnosed cancer in children. T-cell Acute Lymphoblastic Leukemia (T-cell ALL) accounts for around 10-15% of pediatric ALL cases and 25% of adult cases. Constitutive activation of Notch signaling pathway takes place, as a result of NOTCH1 activating mutations, in over 60 % of T-ALL cases. Despite the remarkable improvement that the world has witnessed over the last half-century in the prognosis of ALL, relapse is still a major challenge. Also, the increasing percentage of patients suffering from treatment-related toxicities or chemotherapeutic resistance and recurrence has indicated the inaccuracy of the currently used risk allocation schemes, which consequently explains the necessity of a more reliable assessment approach of the leukemic status. Consequently, a deeper understanding of T-ALL biology at the molecular level is urgently needed to facilitate the identification of novel diagnostic and prognostic biomarkers and therapeutic targets that can ultimately enable early detection, targeted therapy with minimal side effects, and hence a better prognosis. More attention has been paid towards investigating the direct and indirect regulatory roles that NOTCH1 plays in order to promote leukemogenesis. The oncogenic microRNA miR-21 has been implicated in several hematological malignancies and solid tumors. Its oncogenic role in T-ALL is being exhibited through targeting the tumorsuppressive programmed cell death protein 4 (PCDP4). Its aberrant expression has also been associated with a poor prognosis. On the other hand, miR-193b-3p, or miR-193b, is a wellcharacterized tumor-suppressive microRNA that is usually downregulated in a wide range of cancers. In T{u2010} cell acute lymphoblastic leukemia, miR{u2010} 193b{u2010} 3p was also found to be repressed, and its repression was negatively associated with the proto{u2010} oncogene MYB expression. Its downregulation was also found to be associated with unfavorable prognostic features. Although the Notch-regulated long non-coding RNA, LUNAR1, has been demonstrated to be among the most prominently dysregulated lncRNAs in T-ALL cases, the association of its expression levels in the peripheral blood of patients with the clinicopathological features and/or prognosis has not been assessed yet
530 ## - ADDITIONAL PHYSICAL FORM AVAILABLE NOTE
Issues CD Issued also as CD
546 ## - LANGUAGE NOTE
Text Language Text in English and abstract in Arabic & English.
650 #0 - SUBJECT ADDED ENTRY--TOPICAL TERM
Topical term or geographic name entry element Bioechnology
653 #1 - INDEX TERM--UNCONTROLLED
Uncontrolled term ALL
-- MiR-21
-- NOTCH1
700 0# - ADDED ENTRY--PERSONAL NAME
Personal name Mahfouz Ali Abdelaziz
Relator term thesis advisor.
700 0# - ADDED ENTRY--PERSONAL NAME
Personal name Nashwa Nagy Elkhazragy
Relator term thesis advisor.
900 ## - Thesis Information
Grant date 01-01-2022
Supervisory body Mahfouz Ali Abdelaziz
-- Nashwa Nagy Elkhazragy
Universities Cairo University
Faculties Faculty of Science
Department Department of Biotechnology
905 ## - Cataloger and Reviser Names
Cataloger Name Shimaa
Reviser Names Cataloger
942 ## - ADDED ENTRY ELEMENTS (KOHA)
Source of classification or shelving scheme Dewey Decimal Classification
Koha item type Thesis
المقتنيات
Source of classification or shelving scheme Home library Current library Date acquired Inventory number Full call number Barcode Date last seen Effective from Koha item type Copy number
Dewey Decimal Classification المكتبة المركزبة الجديدة - جامعة القاهرة قاعة الرسائل الجامعية - الدور الاول 11.02.2024 85504 Cai01.12.25.M.Sc.2022.Ma.I 01010110085504000 22.09.2023 22.09.2023 Thesis  
Dewey Decimal Classification المكتبة المركزبة الجديدة - جامعة القاهرة مخـــزن الرســائل الجـــامعية - البدروم 11.02.2024 13822081 Cai01.12.25.M.Sc.2022.Ma.I 01020110085504000 22.09.2023 22.09.2023 CD - Rom 85504.CD
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