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Possible modulation of inflammator mediatiors and hepatic fibrosis by an angiotensin II receptor blocker (ARB) in a rat model of CC14-induced liver injury / Sara Abdelmgeed Hamed Elwakeel ; Supervised Ezz Eldin S. Eldenshary , Moushira Ibrahim Elsayed , Noha N. Nassar

By: Contributor(s): Material type: TextLanguage: English Publication details: Cairo : Sara Abdelmgeed Hamed Elwakeel , 2013Description: 90 P. : charts , facsimiles ; 25cmOther title:
  • التغيرات التى يمكن حدوثها للوسطاء الالتهابية والتليف الكبدى باستخدام مثبطات مستقبلات الانجيوتنسين 2 فى نموذج للجرذان المصابة بتليف كبدى ناجم عن استخدام رابع كلوريد الكربون [Added title page title]
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Dissertation note: Thesis (M.Sc.) - Cairo University - Faculty of Pharmacy - Department of Pharmacology and Toxicology Summary: Hepatic fibrosis is defined as a wound healing response to liver injury characterized by the deposition of extracellular matrix (ECM). The duration of insult is a crucial parameter managing the extent of injury. Whereas an acute insult provokes transient changes and the normal hepatic architecture is regenerated, a more persistent injury results in sustained chronic inflammation and accumulation of ECM leading to progressive substitution of liver parenchyma by scar tissue
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Thesis قاعة الرسائل الجامعية - الدور الاول المكتبة المركزبة الجديدة - جامعة القاهرة Cai01.08.09.M.Sc.2013.Sa.P (Browse shelf(Opens below)) Not for loan 01010110062225000
CD - Rom مخـــزن الرســائل الجـــامعية - البدروم المكتبة المركزبة الجديدة - جامعة القاهرة Cai01.08.09.M.Sc.2013.Sa.P (Browse shelf(Opens below)) 62225.CD Not for loan 01020110062225000

Thesis (M.Sc.) - Cairo University - Faculty of Pharmacy - Department of Pharmacology and Toxicology

Hepatic fibrosis is defined as a wound healing response to liver injury characterized by the deposition of extracellular matrix (ECM). The duration of insult is a crucial parameter managing the extent of injury. Whereas an acute insult provokes transient changes and the normal hepatic architecture is regenerated, a more persistent injury results in sustained chronic inflammation and accumulation of ECM leading to progressive substitution of liver parenchyma by scar tissue

Issued also as CD

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