Study of Tumor Necrosis Factor-Related Apoptosis Inducing Ligand (TRAIL) mRNA Expression in Patients with Systemic Lupus Erythematosus/ Mona Mohsen Ahmed Abouelghar ; Supervised Safaa Mostafa Elkaraksy , Noha Mohamed Hosny Shahin , Naglaa Mohamed Kholoussi

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Mona Mohsen Ahmed Abouelghar

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TextLanguage: English Publication details: Cairo : Mona Mohsen Ahmed Abouelghar , 2014Description: 88 P. : charts , facsimiles ; 25cmOther title:

دراسة التعبير للحمض النووي الريبوسومى المرسل لعامل النخر للأورام المرتبط بمحفز موت الخلايا فى مرضى الذئبة الحمراء [Added title page title]

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Dissertation note: Thesis (Ph.D.) - Cairo University - Faculty of Medicine - Department of Clinical and Chemical Pathology Summary: TRAIL (APO-2 ligand) is a transmembrane (type II) glycoprotein which also belongs to the TNF superfamily. TRAIL triggers apoptosis through interaction with the death receptors Death Receptor (DR4) (TRAIL-R1) and (DR5) (TRAIL-R2). There are several indications that TRAIL could be involved in the pathophysiology of autoimmune diseases in general and systemic lupuserythematosus (SLE) in particular. The aim of this study was to investigate TRAIL m RNA expression levels in peripheral blood mononuclear cells(PBMCs) from patients with SLE and to study the association between the results and various clinical and laboratory data of the patients in order to assess the possible role of TRAIL in SLE pathogenesis and its relation to disease activity

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Item type	Current library	Home library	Call number	Copy number	Status	Barcode
Thesis	قاعة الرسائل الجامعية - الدور الاول	المكتبة المركزبة الجديدة - جامعة القاهرة	Cai01.11.07.Ph.D.2014.Mo.S (Browse shelf(Opens below))		Not for loan	01010110063381000
CD - Rom	مخـــزن الرســائل الجـــامعية - البدروم	المكتبة المركزبة الجديدة - جامعة القاهرة	Cai01.11.07.Ph.D.2014.Mo.S (Browse shelf(Opens below))	63381.CD	Not for loan	01020110063381000

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Thesis (Ph.D.) - Cairo University - Faculty of Medicine - Department of Clinical and Chemical Pathology

TRAIL (APO-2 ligand) is a transmembrane (type II) glycoprotein which also belongs to the TNF superfamily. TRAIL triggers apoptosis through interaction with the death receptors Death Receptor (DR4) (TRAIL-R1) and (DR5) (TRAIL-R2). There are several indications that TRAIL could be involved in the pathophysiology of autoimmune diseases in general and systemic lupuserythematosus (SLE) in particular. The aim of this study was to investigate TRAIL m RNA expression levels in peripheral blood mononuclear cells(PBMCs) from patients with SLE and to study the association between the results and various clinical and laboratory data of the patients in order to assess the possible role of TRAIL in SLE pathogenesis and its relation to disease activity

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