FGF23 in incident haemodialysis patients, levels and correlation with vascular calcification / Wessam Abdelrahman Sherif ; Supervised Usama Abdalazeem Sharaf Aldeen , Noha Adel Ibrahim Osman , Mohammed Mahmoud Hassan Elsebaie
Material type:
- عامل النمو الليفى 23 فى المرضى الذين التحقوا حديثاً ببرنامج الإستصفاء الدموى و علاقته بتكلس الأوعية الدموية [Added title page title]
- Issued also as CD
Item type | Current library | Home library | Call number | Copy number | Status | Barcode | |
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قاعة الرسائل الجامعية - الدور الاول | المكتبة المركزبة الجديدة - جامعة القاهرة | Cai01.11.18.M.Sc.2014.We.F (Browse shelf(Opens below)) | Not for loan | 01010110071294000 | ||
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مخـــزن الرســائل الجـــامعية - البدروم | المكتبة المركزبة الجديدة - جامعة القاهرة | Cai01.11.18.M.Sc.2014.We.F (Browse shelf(Opens below)) | 71294.CD | Not for loan | 01020110071294000 |
Thesis (M.Sc.) - Cairo University - Faculty of Medicine - Department of Internal Medicine
During the past decade many factors such as age, systolic blood pressure, hyperphosphatemia and fibroblast growth factor 23 (FGF23) have been addressed as causes of vascular calcification and hence of increasing morbidity and mortality among haemodialysis patients. FGF23, a phosphaturic factor produced by osteoblasts has emerged as a major regulator of mineral metabolism in health and disease. Under physiological conditions, it acts as a circulating phosphaturic factor through suppression of proximal tubular Na / Pi-2c co transporters. FGF23 together with its cofactor Klotho have been found to be expressed and FGF23 to be elevated in patients on haemodialysis
Issued also as CD
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