| 000 | 01679cam a2200265 a 4500 | ||
|---|---|---|---|
| 003 | EG-GiCUC | ||
| 005 | 20250223030221.0 | ||
| 008 | 100420s2009 ua dh f m 000 0 eng d | ||
| 040 |
_aEG-GiCUC _beng _cEG-GiCUC |
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| 041 | 0 | _aeng | |
| 049 | _aGift | ||
| 097 | _aM.Sc | ||
| 099 | _aCai01.34.M.Sc.2009.Ma.N | ||
| 100 | 0 | _aMai Ahmed Saad Zaghloul | |
| 245 | 1 | 0 |
_aNovel anticancer compounds as efficient apoptotic and potential anti - proliferative drugs in genetically modified hepatocellular carcinoma cell models / _cMai Ahmed Saad Zaghloul ; Supervised Ahmed Ihab Abdelaziz |
| 260 |
_aCairo : _bMai Ahmed Saad Zaghloul , _c2009 |
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| 300 |
_a78 Leaves : _bcharts , facsimiles ; _c30cm |
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| 502 | _aThesis (M.Sc.) - German University - Faculty of Postgraduate Studies and Scientific Research - Department of Molecular Genetics | ||
| 520 | _aHepatocellular Carcinoma is one of the severe malignancies with high incidence worldwide as reported to be ranked the sixth most common cancers[2] . Several studies have investigated the underlying pathogenesis of the disease and hypothesized that the interaction between the etiological factors (e.g. HCV, HBV, AFB and cirrhosis [7] and the hepatocytic genes result in functional (imbalance between pro - apoptotic and anti - apoptotic signaling) and structural genomic alterations(loss and gain of chromosomal regions resulting in deletion of tumor suppressor genes e.g, p53 and Rb genes) constituting the basis for HCC pathogenesis [8,44,189] | ||
| 700 | 0 |
_aAhmed Ihab Abdelaziz , _esupervisor |
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| 856 | _uhttp://172.23.153.220/th.pdf | ||
| 905 |
_aFatma _eCataloger |
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| 905 |
_aNazla _eRevisor |
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| 942 |
_2ddc _cTH |
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| 999 |
_c30054 _d30054 |
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