| 000 | 01752cam a2200349 a 4500 | ||
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| 003 | EG-GiCUC | ||
| 005 | 20250223030654.0 | ||
| 008 | 120917s2012 ua dh f m 000 0 eng d | ||
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_aEG-GiCUC _beng _cEG-GiCUC |
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| 041 | 0 | _aeng | |
| 049 | _aDeposite | ||
| 097 | _aPh.D | ||
| 099 | _aCai01.11.07.Ph.D.2012.Al.A | ||
| 100 | 0 | _aAlaa Afif Abdelraouf | |
| 245 | 1 | 4 |
_aThe association between kir6.2 mutation and type 1 diabetes / _cAlaa Afif Abdelraouf ; Supervised Nadida Abdelhamid Gohar , Shereen Abdelghaffar Taha , Nahla Aly Fawzy Fayek |
| 246 | 1 | 5 | _aالعلاقة بين التحور الجينى للجين ( كير 2و6 ) والنوع الاول من البول السكرى |
| 260 |
_aCairo : _bAlaa Afif Abdelraouf , _c2012 |
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| 300 |
_a81Leaves : _bcharts , facsimiles ; _c25cm |
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| 502 | _aThesis (Ph.D.) - Cairo University - Faculty of Medicine - Department of Clinical and Chemical Pathology | ||
| 520 | _aThe pancreatic ATP - sensitive potassium ( Katp ) channel is a critical regulator of beta - cell insulin secretion . Heterozygous activating mutations in the KCNJ11 gene , which encodes the Kir 6.2 subunit of the Katp channel , have been found to cause diabetes in the neonatal period or early infancy . This molecular study hypothesized that KCNJ11 mutations could present clinically as type 1 diabetes | ||
| 530 | _aIssued also as CD | ||
| 653 | 4 | _aKatp channel | |
| 653 | 4 | _akir6.2 | |
| 653 | 4 | _aNeonatal diabetes | |
| 700 | 0 |
_aNadida Abdelhamid Gohar , _eSupervisor |
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| 700 | 0 |
_aNahla Aly Fawzy Fayek , _eSupervisor |
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| 700 | 0 |
_aShereen Abdelghaffar Taha , _eSupervisor |
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| 856 | _uhttp://172.23.153.220/th.pdf | ||
| 905 |
_aFatma _eCataloger |
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| 905 |
_aNazla _eRevisor |
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| 942 |
_2ddc _cTH |
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| 999 |
_c39529 _d39529 |
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