| 000 | 02123cam a2200349 a 4500 | ||
|---|---|---|---|
| 003 | EG-GiCUC | ||
| 005 | 20250223030943.0 | ||
| 008 | 140317s2013 ua dh f m 000 0 eng d | ||
| 040 |
_aEG-GiCUC _beng _cEG-GiCUC |
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| 041 | 0 | _aeng | |
| 049 | _aDeposite | ||
| 097 | _aM.Sc | ||
| 099 | _aCai01.08.09.M.Sc.2013.Sa.P | ||
| 100 | 0 | _aSara Abdelmgeed Hamed Elwakeel | |
| 245 | 1 | 0 |
_aPossible modulation of inflammator mediatiors and hepatic fibrosis by an angiotensin II receptor blocker (ARB) in a rat model of CC14-induced liver injury / _cSara Abdelmgeed Hamed Elwakeel ; Supervised Ezz Eldin S. Eldenshary , Moushira Ibrahim Elsayed , Noha N. Nassar |
| 246 | 1 | 5 | _aالتغيرات التى يمكن حدوثها للوسطاء الالتهابية والتليف الكبدى باستخدام مثبطات مستقبلات الانجيوتنسين 2 فى نموذج للجرذان المصابة بتليف كبدى ناجم عن استخدام رابع كلوريد الكربون |
| 260 |
_aCairo : _bSara Abdelmgeed Hamed Elwakeel , _c2013 |
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| 300 |
_a90 P. : _bcharts , facsimiles ; _c25cm |
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| 502 | _aThesis (M.Sc.) - Cairo University - Faculty of Pharmacy - Department of Pharmacology and Toxicology | ||
| 520 | _aHepatic fibrosis is defined as a wound healing response to liver injury characterized by the deposition of extracellular matrix (ECM). The duration of insult is a crucial parameter managing the extent of injury. Whereas an acute insult provokes transient changes and the normal hepatic architecture is regenerated, a more persistent injury results in sustained chronic inflammation and accumulation of ECM leading to progressive substitution of liver parenchyma by scar tissue | ||
| 530 | _aIssued also as CD | ||
| 653 | 4 | _aAngiotensin II | |
| 653 | 4 | _aHepatic fibrosis | |
| 653 | 4 | _aLiver injury | |
| 700 | 0 |
_aEzz Eldin S. Eldenshary , _eSupervisor |
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| 700 | 0 |
_aMoushira Ibrahim Elsayed , _eSupervisor |
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| 700 | 0 |
_aNoha N. Nassar , _eSupervisor |
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| 856 | _uhttp://172.23.153.220/th.pdf | ||
| 905 |
_aAml _eCataloger |
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| 905 |
_aNazla _eRevisor |
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| 942 |
_2ddc _cTH |
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| 999 |
_c45419 _d45419 |
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