000			02358cam a2200349 a 4500
003			EG-GiCUC
005			20250223032317.0
008			190619s2019 ua d f m 000 0 eng d
040			_aEG-GiCUC _beng _cEG-GiCUC
041	0		_aeng
049			_aDeposite
097			_aPh.D
099			_aCai01.11.09.Ph.D.2019.Mo.E
100	0		_aMohamed Ahmed Obaya
245	1	0	_aEarly versus delayed administration of norepinephrine in patients with septic shock and use of oral midodrine in weaning off norepinephrine in these patients / _cMohamed Ahmed Obaya ; Supervised Alia Abdelfattah , Randa Ali , Moemen Yehia
246	1	5	_aإعطاء النورادرينالين فى وقت مبكر مقابل تأخر اعطاؤه لمرضى الصدمة الإنتانية وأستخدام الميدودرين بالفم فى الفطام من النورادرينالين فى هؤلاء المرضى
260			_aCairo : _bMohamed Ahmed Obaya , _c2019
300			_a123 P. : _bcharts ; _c25cm
502			_aThesis (Ph.D.) - Cairo University - Faculty of Medicine - Department of Critical Care Medicine
520			_aedefine sepsis as agreed upon by The Society of Critical Care Medicine (SCCM) and the European Society of Intensive Care Medicine (ESICM) as the Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3) (Singer M., et al. 2016). Sepsis: 2life-threatening organ dysfunction caused by a dysregulated host response to infection.3 End organ damage is identified as an acute change in total Sequential [Sepsis-related] Organ Failure Assessment score (SOFA) {u2265}2 (Rhodes A., et al 2016). Septic shock: A subset of sepsis 2in which circulatory, cellular, and metabolic abnormalities are associated with a greater risk of mortality than with sepsis alone. These patients can be clinically identified by a vasopressor requirement to maintain a MAP {u2265} 65mmHg and serum lactate >2mmol/L in the absence of hypovolemia (Singer M., et al 2016)
530			_aIssued also as CD
653		4	_aMidodrine
653		4	_aNorepinephrine
653		4	_aSeptic Shock
700	0		_aAlia Abdelfattah , _eSupervisor
700	0		_aMoemen Yehia , _eSupervisor
700	0		_aRanda Ali , _eSupervisor
856			_uhttp://172.23.153.220/th.pdf
905			_aAsmaa _eCataloger
905			_aNazla _eRevisor
942			_2ddc _cTH
999			_c72468 _d72468