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040 _aEG-GiCUC
_beng
_cEG-GiCUC
041 0 _aeng
049 _aDeposite
097 _aM.Sc
099 _aCai01.08.09.M.Sc.2021.Me.P
100 0 _aMerhan Ossama Abdelghany Hindam
245 1 0 _aPossible neuroprotective effects of the coumarins :
_bXanthotoxin and umbelliferone in the model of streptozotocin-induced sporadic Alzheimer{u2019}s disease in rats /
_cMerhan Ossama Abdelghany Hindam ; Supervised Nesrine S. Elsayed , Rabab Hamed Sayed
246 1 5 _aزانثوتوكسين و امبيلليفرون في النموذج المحدث به مرض الزهايمر المنتشر بفعل استربتوزوتوسين في الجرذان :
_bالتأثيرات الممكنة الوقائية للأعصاب لكومارينز
260 _aCairo :
_bMerhan Ossama Abdelghany Hindam ,
_c2021
300 _a148 P . :
_bcharts , facsmilies ;
_c25cm
502 _aThesis (M.Sc.) - Cairo University - Faculty of Pharmacy - Department of Pharmacology and Toxicology
520 _aThe aim of the present study was to assess the neuroprotective effects of xanthotoxin and umbelliferone in streptozotocin (STZ)-induced cognitive dysfunction in rats. Animals were injected intracerebroventricularly (ICV) with STZ (3 mg/kg) once to induce a sporadic Alzheimer's disease (SAD)-like condition. Xanthotoxin or umbelliferone (15 mg/kg, i.p.) were administered 5 hr after ICV-STZ and daily for 20 consecutive days. Xanthotoxin or umbelliferone prevented cognitive deficits in the Morris water maze and object recognition tests. In parallel, xanthotoxin or umbelliferone reduced hippocampal acetylcholinestrase activity and malondialdehyde level. Moreover, xanthotoxin or umbelliferone increased glutathione content. These coumarins also modulated neuronal cell death by reducing the level of proinflammatory cytokines) tumour necrosis factor-alpha and interleukin-6), inhibiting the overexpression of inflammatory markers (nuclear factor mB [NF-mB] and cyclooxygenase II), and upregulating the expression of NF-mB inhibitor (ImB-Ü). Interestingly, xanthotoxin diminished phosphorylated JAK2 and phosphorylated STAT3 protein expression, while umbelliferone markedly replenished nuclear factor erythroid-derived 2-like 2 (Nrf2) and haem oxygenase-1 (HO-1) levels. The current study provides evidence for the protective effect of xanthotoxin and umbelliferone in STZ-induced cognitive dysfunction in rats. This effect may be attributed, at least in part, to inhibiting acetylcholinestrase and attenuating oxidative stress, neuroinflammation and neuronal loss.
530 _aIssued also as CD
653 4 _aAlzheimer's disease
653 4 _aUmbelliferone
653 4 _aXanthotoxin
700 0 _aNesrine S. Elsayed ,
_eSupervisor
700 0 _aRabab Hamed Sayed ,
_eSupervisor
856 _uhttp://172.23.153.220/th.pdf
905 _aAmira
_eCataloger
905 _aNazla
_eRevisor
942 _2ddc
_cTH
999 _c80650
_d80650