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040 _aEG-GiCUC
_beng
_cEG-GiCUC
041 0 _aeng
_beng
_bara
049 _aDeposite
082 0 4 _a632.9517
092 _a632.9517
_221
097 _aPh.D
099 _aCai01.16.03.Ph.D.2022.He.E
100 0 _aHeba Moustafa Rasheed Hathout.
_epreparation.
245 1 0 _a Evaluation of the genotoxicity and oxidative stress of acetamiprid insecticide on nile tilapia :
_bOreochromis niloticus /
_cHeba Moustafa Rasheed Hathout; Supervised Prof. Dr. Hassan. M. Sobhy
246 1 5 _aتقييم السمية الجينية والجهد التأكسدي للمبيد الحشري اسيتامبريد على البلطي النيلي
264 0 _c2022
300 _a123p. :
_bcharts, facsimiles ;
_c25cm.
_eCD
336 _atext
_2rda content
337 _aUnmediated
_2rdamedia
338 _avolume
_2rdacarrier
502 _aThesis (Ph.D.) - Cairo University - Faculty of African Postgraduate Studies - Department of Natural Resources
504 _aBibliography: p. 123-98.
520 3 _aAcetamiprid (Aceta) is the most extensively used neonicotinoid which contaminate aquatic ecosystem from agricultural leaching. On juveniles of Oreochromis niloticus, the protective potential of ascorbic acid (Asc) against oxidative stress and genotoxicity induced by Aceta sub-lethal concentrations was investigated in this study. Fishes were divided into six groups and exposed to either; Asc (50 ppm), 10 ppm of Aceta, 20 ppm of Aceta, 10 ppm of Aceta and Asc (50 ppm), 20 ppm of Aceta and Asc (50 ppm). Unexposed animals were considered as control group. The experiment was conducted by the assessment of superoxide dismutase (SOD), catalase (CAT) and glutathione peroxidase (GPX) activities and their mRNA gene expression. DNA damage using comet assay in erythrocytes, hepatocytes and gill cells, in addition to mitotic index (MI), the existence of micronuclei (MN) and erythrocytic nuclear abnormalities (ENAs) were performed. The results showed that concentrations of Aceta (10 & 20 ppm) induced oxidative stress by decreasing the antioxidant enzyme activities and down-regulating gene expression of SOD, CAT and GPX compared to control group. However, LPO and NO levels were significantly elevated. Co-supplemented fish with Asc showed significant alleviations of oxidative status represented by substantial increases in the activities of SOD, CAT and GPX enzymes and diminished LPO and NO levels (P<0.05), along with a concomitant up-regulation of SOD and CAT gene expression (P < 0.05). There were genotoxic effects of Aceta exposure showed by the significant (P < 0.05) increase in DNA-damaged cells, MN, and ENA, meanwhile a decrease in MI. Supplementation with Asc reversed these effects in combined-exposure groups. Thus, results suggest that Aceta induced oxidative stress accompanied with genotoxicity. Further, Asc-combined exposure could be the effective treatment against Aceta-induced damage in O. niloticus.
520 3 _aأعدت هذه الدراسة لفحص القدرة الوقائية لحمض الأسكوربيك ضد الجهد التأكسدي والسمية الجينية المسببة بواسطة جرعات المبيد الحشري (إسيتامبريد على صغار أسماك البلطي النيلي.
530 _aIssued also as CD
546 _aText in English and abstract in Arabic & English.
650 0 _aGenotoxicity
653 1 _aAcetamiprid Insecticide
_aNile Tilapia
_aOxidative Stress
700 0 _aHassan. M. Sobhy
_ethesis advisor.
700 0 _aIslam M. El-Garawani
_ethesis advisor.
900 _b01-01-2022
_cHassan. M. Sobhy
_UCairo University
_FFaculty of African Postgraduate Studies
_DDepartment of Natural Resources
905 _aShimaa
942 _2ddc
_cTH
999 _c84718