Role of single nucleotide polymorphism in DNA repair genes in cytogenetic status in acute myeloid leukemia patients / Mahmoud Aly Mohamed Ayoub ; Supervised Farida Hassan Gadallah , Gamal Thabet Ebid , Essam Hamed Elnoshokaty

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Mahmoud Aly Mohamed Ayoub

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TextLanguage: English Publication details: Cairo : Mahmoud Aly Mohamed Ayoub , 2014Description: 114 P. : charts , facsimiles ; 25cmOther title:

العلاقة بين الجينات المسئولة عن إصلاح الحمض النووى و مدى تأثيرها على التشوهات الوراثية لسرطان الدم الميلودى الحاد [Added title page title]

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Dissertation note: Thesis (Ph.D.) - Cairo University - National Cancer Institute - Department of Clinical Pathology Summary: Acute myeloblastic leukemia (AML) is a clonal disorder characterized by acquisition of somatic mutations in haematopoietic progenitors cells. Defects in DNA repair genes and proteins associated with the regulation of repair are connected to many different human diseases including acute myeloid leukemia. Single nucleotide polymorphisms SNPs in many DNA repair genes revealed to be associated with cytogenetic abnormalities among newly diagnosed AML patients

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Item type	Current library	Home library	Call number	Copy number	Status	Barcode
Thesis	قاعة الرسائل الجامعية - الدور الاول	المكتبة المركزبة الجديدة - جامعة القاهرة	Cai01.19.03.Ph.D.2014.Ma.R (Browse shelf(Opens below))		Not for loan	01010110064497000
CD - Rom	مخـــزن الرســائل الجـــامعية - البدروم	المكتبة المركزبة الجديدة - جامعة القاهرة	Cai01.19.03.Ph.D.2014.Ma.R (Browse shelf(Opens below))	64497.CD	Not for loan	01020110064497000

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Thesis (Ph.D.) - Cairo University - National Cancer Institute - Department of Clinical Pathology

Acute myeloblastic leukemia (AML) is a clonal disorder characterized by acquisition of somatic mutations in haematopoietic progenitors cells. Defects in DNA repair genes and proteins associated with the regulation of repair are connected to many different human diseases including acute myeloid leukemia. Single nucleotide polymorphisms SNPs in many DNA repair genes revealed to be associated with cytogenetic abnormalities among newly diagnosed AML patients

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