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Effect of thymoquinone on Ü7 nicotinic acetylcholine receptor in LPS- induced neuroinflammatory Alzheimer's disease rat model / Esrss Osama Abdelaziz Mostafa ; Supervised Ola Heikal , Lobna Abdelaal Kassem

By: Contributor(s): Material type: TextTextLanguage: English Publication details: Cairo : Esrss Osama Abdelaziz Mostafa , 2017Description: 139 Leaves : charts , facsimiles ; 30cmOnline resources: Dissertation note: Thesis (M.Sc.) - German University - Faculty of Postgraduate Studies and Scientific Research - Department of Pharmacology and toxicology Summary: Alzheimer's disease (AD) is a neurodegenerative disorder resulting from the loss of cholinergic nerurons in the brain. It has been reported that Ü7 nicotinic acetylcholine receptors(Ü7 nAChRs) play an important role in cognitive processes representing a durg target for treating congnitive deficits. Ü7 nAChRs agonists cause memory enhancement through phosphorylation of cAMP response element binding protein (CREB)
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Thesis Thesis قاعة الثقاقات الاجنبية - الدور الثالث المكتبة المركزبة الجديدة - جامعة القاهرة Cai01.34.M.Sc.2017.Es.E (Browse shelf(Opens below)) Not for loan 01010110075323000

Thesis (M.Sc.) - German University - Faculty of Postgraduate Studies and Scientific Research - Department of Pharmacology and toxicology

Alzheimer's disease (AD) is a neurodegenerative disorder resulting from the loss of cholinergic nerurons in the brain. It has been reported that Ü7 nicotinic acetylcholine receptors(Ü7 nAChRs) play an important role in cognitive processes representing a durg target for treating congnitive deficits. Ü7 nAChRs agonists cause memory enhancement through phosphorylation of cAMP response element binding protein (CREB)

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