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Serum and tissue 2HVEM3 levels in patients with multiple or recurrent verruca vulgaris / Marwa Abdelghany Mansour ; Supervised Manal A-W Bosseila , Olfat G. Shaker , Dina M. Kadry

By: Contributor(s): Material type: TextTextLanguage: English Publication details: Cairo : Marwa Abdelghany Mansour , 2015Description: 114 P. : charts ; 25cmOther title:
  • مستوى مادة "الهيفم " في المصل والجلد للمرضى المصابين بالثآليل الفيروسية المتعددة أو المتكررة [Added title page title]
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  • Issued also as CD
Dissertation note: Thesis (M.Sc.) - Cairo University - Faculty of Medicine - Department of Dermatology and Venereology Summary: Warts are benign lesions caused by human papilloma virus (HPV). Host defense against HPV relies on intact and functioning cellular immunity including T cell and natural killer (NK) cell cytotoxicity. Therefore, in patients in whom warts are severe, concern for immune defects is raised. HVEM (herpes virus entry mediator) is a member of the tumour-necrosis factor receptor family. It has diverse functions, augmenting or inhibiting the immune response. It acts as a receptor to enhance immune responses through binding LIGHT or LTalpha which signaling is important for cell survival, inflammation, and tumor eradication. {uF0A7}Aim of work: To identify the role of cell-mediated immunity, expressed as HVEM level, in cases of multiple/recurrent verrucae, in order to assess its role in the adaptive immune response to HPV
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Item type Current library Home library Call number Copy number Status Barcode
Thesis Thesis قاعة الرسائل الجامعية - الدور الاول المكتبة المركزبة الجديدة - جامعة القاهرة Cai01.11.10.M.Sc.2015.Ma.S (Browse shelf(Opens below)) Not for loan 01010110069070000
CD - Rom CD - Rom مخـــزن الرســائل الجـــامعية - البدروم المكتبة المركزبة الجديدة - جامعة القاهرة Cai01.11.10.M.Sc.2015.Ma.S (Browse shelf(Opens below)) 69070.CD Not for loan 01020110069070000

Thesis (M.Sc.) - Cairo University - Faculty of Medicine - Department of Dermatology and Venereology

Warts are benign lesions caused by human papilloma virus (HPV). Host defense against HPV relies on intact and functioning cellular immunity including T cell and natural killer (NK) cell cytotoxicity. Therefore, in patients in whom warts are severe, concern for immune defects is raised. HVEM (herpes virus entry mediator) is a member of the tumour-necrosis factor receptor family. It has diverse functions, augmenting or inhibiting the immune response. It acts as a receptor to enhance immune responses through binding LIGHT or LTalpha which signaling is important for cell survival, inflammation, and tumor eradication. {uF0A7}Aim of work: To identify the role of cell-mediated immunity, expressed as HVEM level, in cases of multiple/recurrent verrucae, in order to assess its role in the adaptive immune response to HPV

Issued also as CD

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